We have a 2 year old son that has rae things going on with him and we are in deep search of to find a way to help him. If reading through this and you see a way you may help in any way please contact us. We are trying to make life easier for our son and other children as well.

Thank you so much from a family with hope~~~~~~~

HISTORY

Tanner was born at 39 weeks by C-section due to Mom having previous C-sects.His apgar score was a 9. At birth he was slightly jaundiced.His weight was 7 lbs.8 oz..His length was 19.5 inches. Mom was concerned about his eyes as they seemed to disapear downwards more often than not and long time intervels.

7-2-02 admitted to mckay dee hospital Ogden,Utah. For fevers....meningitis..heart murmur...

labs....4 spinal taps all cloudy one with clots....shown RBC and WBC elevated

high calcium levels

7-6-02 Discharged with viral meninitis

7-9-02 admitted back at Mckay dee

medicines given at this time----ampicillin,gentamicin,and acylovir at encephlitis/meningitis dosage

7-12-02 discharged

7-?-02 was put on apnea monitor

8-7-02 Saw DR.swinyard (endocrinologist) thought he has hyperparathyroidism later changed to hypercalcemia

8-30-02 PH probe study done to rule out reflux as cause of apnea----reflux present but not cause of apnea

9-26-02 CT-scan Impression as follows:Mild to moderate enlargement of the lateral ventricles particularly to the left.

10-5-02 MRI of brain without contrast

Impression as follows: Widening of the sylvian fissures and hypoplastic development of the temporal lobes and opercular frontal lobes can be associated with an organic acid abnormality.recommend a metabolic work up.

Mild dilation of the lateral ventricles perhaps from exvacuo dilation.

Mild Chiari I Malformation with the tonsild extending through the foramen magnum. Normal CSF flow. Incidental note of a probable mega cisterna magna vs. a arachnoid cyst in the poster fossa.

4-02 to 10 -02 Weight and hieght plateued put on pediasure three times a day

7-24-03 stopped breathing passed out was resiucitated was pale and week for a half hour afterwards believed seizure and postictal state.

12-3-03 follow up MRI performed

Impression as follows:: Follow up examination continues to show lateral ventricular dilation with probable slight progression and interim development of abnormal signal and periventricular white matter.Pattern nonspecific but possibility of degenerative white matter disease questioned. No other change.

1-10-04 that week Taner had blue spells He turned black-his arms legs and face----At times he would breathe rapidly his heart would race..Was in Mckay Dee for a breif visit hada CT scan done at this time.

6-20-04 Started limping wouldnt walk on one foot right fell often.

6-21-04 doctors did Xrays blood work found nothing believe it is ataxia due to neurological probs. Cant get his neuro team to see him till sept.

6-23-04 still his leg isnt doing well notice his hand opposite side is being held funny

* note his first year of life he screamed always as if in intense pain.Found taht music with the bass turned way up and him laying next to the speakers would help him sleep if turned off he would wake with the same scream.

MRI 1

MRI BRAIN W/O CNT 05 OCT 02 10:00

MILD VENTRICULAR ENLARGEMENT/A

Exam:MRI ofthe brain without contrast dated 10/5/02 at 1005.

oral sedation

Comparison: No

History: apnea and bradycardia, ventriculomegaly

Sedation: After a directed history and physical examination, the patient was sedated using oral Nembutal, monitored by the sedation nurse in the standard fashion without immediate complication

Technique: Sagittal T1 , axial PD/T2/FLAIR, coronal /sagittal T2 Cine pahse contrast imaging through the skull base.

FINDINGS: There is symmetric atrophy adn hypoplastic development of the temporal lobes bilaterally. There is as well hypoplasia of the frontal lobes adjacent to the sylvian fissures.

The lateral ventricles are meildly enlarged. The third and fourth ventricles are within normal limits. Extra-axial fluid spaces are otherwise normal.

There is no evidence for cortical dysplasia. The brainstem and posterior fossa contents are within normal limits. The cerebellar tonsils extend just through foramen magnum with normal CSF flow. The mylination pattern is within normal limits for age.

IMPRESSION: Widening of the sylvian fissures and hyploplastic development of the temporal lobes can be associated with an organic acid abnormality.Recommen a metabolic work-up

Mild dilation of the lateral ventricles perhaps from ex vacuo dialtion.

Mild Chairi I malformation with the tonsils extending thrugh the foramen magnum. normal CSf flow. Incidental note of a probable mega cisterna magna vs. a arachnoid cyst in the poster fossa.

MRI 2

NAME: WINES.TANNER RYAN

EXAM: MRI BRAIN W/O CNT

RSN1:ENLARGED VENTS/CHIARI MALFORMATION

RSN2:UNAVAILABLE

BRAIN MRI WITH SEDATION. COMPARISON 10.5.02

History : developmental delay ,apnea,seizures, hydrocephalous

Technique and findings: I performed a pre-sedation history and physical examination and suprvised intravenous sedtion. Continous monitoring done by sedation nursing personnel through procedure and during recovery. No adverse response to sedation noted.Multi-planar imaging done using diffrering magnetic parameters. Spectroscopy performed adn reported seperately.

Moderate lateral ventricular dialtion. Third and Fourth ventricules are not dialted. Probable slight progressive dialtion of anterior horn portion of lateral ventricles. Progressive myelinization has developed and myelinization is appropriate for age. Small areas of abnormal signal in periventricular white matter. This has progressed since prior study. Otherwise, no abnormal parenchymal signal identified. Extra axial fluid is normal in volume. Mild downaward extension of cerebellar tissue through foramen magnum in which does not appear compressed and is unchanged as compared with previous study and of doubtful significance. effects of apnea with hypoxia cannot be excluded but pattern not typical of this setting.

Impression: Follow-up examination continues to show lateral ventricular dilation with probable slight progression and interim development of abnormal signal and periventricular white matter. Pattern nonspecific but possibility of degenerative white matter disease questioned . no other interval change.

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DOCTORS

The Arachnoid Cyst Foundation
c/o Catherine Clay
PO Box 66820
Los Angeles, Ca 90066
1-(800)-493-5022
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